Type-2 diabetes disease is characterized by the lost of capabilities of the beta cells to segregate granules of insulin. The action of segregation has as previous processes the entrance of Calcium 2+ ions inside the beta cells by passing the voltage-gated channel while is open. We present a scheme where a prospective frequency-based nanodevice deployed in adjacent areas of the Langerhans islets would emit permanently bunches of Calcium 2+. Once that these ions have surpassed the potential membrane acquire position to electrically interact with the granules of insulin so one expects that most of them might be released continuously, thus meanwhile it's done the pre-diabetic patient does not surpass the line that separates the condition of having or not the diagnosis of type-2 diabetes. We present computational simulations that to some extent provide an idea about the effectiveness of this induced mechanism for insulin production as well as we identified limitations on these artificial mechanisms as to produce hyperinsulinism or another anomalous consequence of these electric nanodevices.